index - Organisation de la cellule musculaire et thérapie de la myopathie centronucléaire autosomique dominante Accéder directement au contenu

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Autophagosome AAV8 Cell proliferation AAV Dynamin overexpression DMyHC Centronuclear myopathy Dystrophie musculaire de Duchenne Dynamine Correlative microscopy Caveolins Neural crest cells Disease modifiers Developmental myosin heavy chain Gene therapy Coeur Alpha-actinin-2 Developmental biology Diaphragm AD-CNM Cytoskeleton Duchenne muscular dystrophy Clathrine Cell signaling Muscle Atrial heart defects Charcot-Marie-Tooth Congenital myopathy Outflow tract Cellular neuroscience Atrial cardiac defects Antisense oligonucleotides Autophagy Myopathy Dominant centronuclear myopathy Cardiomyopathies Lamin Dystrophin Endocytosis Duchenne Muscular Dystrophy Dullard Allele specific RNA interference AFM Allele‐specific silencing therapy DNM2 Disease heterogeneity Cross-presentation RNA interference Adeno-Associated virus Nesprin Becker muscular dystrophy BMD Cardiotoxin Core myopathy Domaine LEM Cell migration Nucleus Cellules de crête neurale Biomarkers Biophysics Skeletal muscle ACTN2 Autophagosome maturation Autosomal dominant centronuclear myopathy Animal models of human disease Actin nucleus Cancer BAR proteins Myopathie Cytosquelette Adult patients Migration Satellite cell Adeno-associated virus vector Allele-specific silencing Dynamin Actin Adhesion Myosin Dynamin 2 CAV-3 gene Cavéoles Ctdnep1 CTL Allele-specific silencing therapy Cross-bridge kinetics BMP signaling BAF Nuclear envelope A-type lamins Duchenne muscular dystrophy DMD Amphiphysin Dystrophie musculaire d'Emery Dreifuss Adeno-associated virus Cavins Skin Mechanotransduction Caveolin Caveolae Muscular dystrophy Clathrin